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Exploratory Laparotomy (Emergent)

Patient phenotype

Acute abdomen — perforated viscus, ischemic bowel, ruptured AAA, gunshot/trauma. Often septic, hypovolemic, full stomach. Age varies wildly.

Procedure

Midline incision, exploration, source control. Length highly variable (45 min for perfed appy → 6+ hours for bowel resection + washout). Often needs vasopressor support throughout.

Anesthetic plan

RSI GETA. A-line pre-induction if hemodynamically unstable. CVC if pressors expected to last > 2 hours or need for CVP/ScvO₂. Massive transfusion protocol activated if surgical bleeding likely. Avoid N₂O (bowel distension).

Setup

  • ·2× 16-gauge PIVs minimum, blood tubing primed
  • ·A-line — pre-induction if shock
  • ·CVC if pressors ≥ 2-3 mcg/kg/min equivalent or fluid status unclear
  • ·Type & cross 4 units PRBC, FFP available
  • ·Rapid infuser (Belmont / Level 1) ready
  • ·Forced air warmer + fluid warmer
  • ·Foley with hourly UOP

Biggest concerns by phase

Pre-op

Resuscitation status — under-fluid is more dangerous than over-fluid here

Septic + hypovolemic patient looks borderline OK at rest, then crashes on induction. Goal: at least 30 mL/kg balanced crystalloid before induction unless cardiogenic concern. Lactate, base deficit, MAP trend > one-time vital sign.

Induction

RSI in shock — drug choice + dose reduction

Standard induction doses cause cardiovascular collapse in shock. Etomidate 0.1–0.2 mg/kg or ketamine 0.5–1 mg/kg with sux 1–1.5 mg/kg or roc 1.2 mg/kg. Pre-position vasopressors (phenylephrine, norepinephrine) drawn. Be ready to start NE infusion immediately.

Intra-op

Hypothermia + coagulopathy + acidosis (lethal triad)

Long laparotomy + open viscera + cold IV fluids = rapid heat loss. Aim core temp ≥ 36 °C. Use warm fluids, BAIR all surfaces possible, room temp up. Below 35 °C, platelet function drops, factor activity drops 10% per °C.

Intra-op

Massive transfusion + 1:1:1 ratio

If transfusion exceeds 4 units in 1 hr or 10 units in 24 hr, activate MTP. Give PRBC : FFP : platelets ≈ 1:1:1. Replace cryo if fibrinogen < 200. Avoid LR (calcium chelation by citrate = hypocalcemia + worsened coag).

Intra-op

Hypocalcemia from citrate + transfusion

Each unit of PRBC contains ~3 g citrate. Liver normally clears it; in shock or rapid transfusion the citrate binds calcium → ionized hypocalcemia → cardiac dysfunction, prolonged QT. Replace empirically: CaCl₂ 1 g per 4 units PRBC.

Emergence

Don't extubate — go to ICU intubated

Even if vitals look 'OK,' the patient resuscitated through major surgical insult is at high risk for re-deterioration. Standard practice: ICU intubated overnight, wean per ICU protocol after sedation and pressor wean. Discussion with surgeon + ICU before emergence.

Mock-defense scenarios

Practice answering these out loud. The probes show what an examiner is listening for.

Your patient is a 65-year-old male with a perforated diverticulum. BP 85/50, HR 120, lactate 6.2, K 5.8, getting his second 30-mL/kg bolus on the way to OR. Surgeon wants to start now. Walk me through induction.

What an examiner probes for
  • Acknowledges resuscitation incomplete + risk vs. surgical urgency
  • Drug choice: etomidate vs. ketamine + dose-reduced + sux/roc rationale
  • Pre-positioned vasopressor + a-line pre-induction
  • K+ + acidosis management (avoid sux? — actually OK at K 5.8 if RSI urgency)

You've given 6 units PRBC, 4 FFP, 1 6-pack platelets. INR is 1.8, fibrinogen 140, ionized Ca 0.9, temp 35.2. Surgeon says they need 'a few more units, we're almost done.' What's your plan?

What an examiner probes for
  • Cryo for fibrinogen < 200
  • CaCl₂ for ionized hypocalcemia
  • Active warming — fluid warmer, BAIR, ambient temp
  • Communicate with surgeon: lethal triad recognized, may need damage control → ICU

Sources

  • Miller's Ch 81 (trauma)
  • ASA MTP guidelines
  • ATLS 10e

Anatomy reference

Sourced reference images. 4 matches for "abdomen peritoneum bowel".

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Education only — anesthetic plans vary by patient, institution, and provider judgment. Use as a starting point, not a substitute for clinical reasoning.